That's what they call it when you are vaccine injured. Her tales of the doctor visits and severe headaches, etc. are associated with vaccine injury.

"Just because your child does not have mitochondrial disorder before being vaccinated does not mean he or she won’t have it after vaccination. That’s because the aluminum used as an adjuvent in vaccines causes mitochondrial disorder. So, if your child is vaccinated on the first day of his or her life with the hepatitis B vaccine, which contains high levels of aluminum, and then you have your child tested for mitochondrial disorder, and the test is positive, there’s no way to know where it came from. Unless you are also tested and you test negative.

Here is some information from a recent article (peer-reviewed medical literature) about aluminum and mitochondrial disorder:

In the present review we describe how the use of a systems biology approach in cultured hepatoblastoma cells (HepG2) allowed the identification of the molecular targets of Al toxicity. Mitochondrial metabolism is the main site of the toxicological action of Al. Fe-dependent and redox sensitive enzymes in the tricarboxylic acid (TCA) cycle and oxidative phosphorylation (OXPHOS) are dramatically decreased by Al exposure. In an effort to compensate for diminished mitochondrial function, Al-treated cells stabilize hypoxia inducible factor -1a (HIF-1a) to increase ATP production by glocolysis. Additinoally, Al toxicity leads to an increase in intracellular lipid accumulation due to enhanced lipogenesisi and a decrease in the B-oxidation of fatty acids. Central to these effects is the alteration of a-ketoglutarate (KG) homeostasis. In Al-exposed cells, KG is preferentially used to quench ROS leading to succinate accumulation and HIF-1a stabilization. Moreover, the channeling of KG to combat oxidative stress leads to a reduction of L-carnitine biosynthesis and a concomitant decrease in fatty acid oxidation. The fluidity and interaction of these mtabolic modules and the implications of these findings in liver-related disorders are discussed herein. (citation) (Note: The importance of these findings is relevant especially for brain development and for chronic autoimmune disorders such as diabetes and autism. The disruption of mitochondrial metabolism is significant in the increased risk of vaccine-injury in persons who have underlying mitochondrial disorders – See the Hannah Poling case for more information.)" Just because your child does not have mitochondrial disorder before being vaccinated does not mean he or she won’t have it after vaccination. That’s because the aluminum used as an adjuvent in vaccines causes mitochondrial disorder. So, if your child is vaccinated on the first day of his or her life with the hepatitis B vaccine, which contains high levels of aluminum, and then you have your child tested for mitochondrial disorder, and the test is positive, there’s no way to know where it came from. Unless you are also tested and you test negative.

Here is some information from a recent article (peer-reviewed medical literature) about aluminum and mitochondrial disorder:

In the present review we describe how the use of a systems biology approach in cultured hepatoblastoma cells (HepG2) allowed the identification of the molecular targets of Al toxicity. Mitochondrial metabolism is the main site of the toxicological action of Al. Fe-dependent and redox sensitive enzymes in the tricarboxylic acid (TCA) cycle and oxidative phosphorylation (OXPHOS) are dramatically decreased by Al exposure. In an effort to compensate for diminished mitochondrial function, Al-treated cells stabilize hypoxia inducible factor -1a (HIF-1a) to increase ATP production by glocolysis. Additinoally, Al toxicity leads to an increase in intracellular lipid accumulation due to enhanced lipogenesisi and a decrease in the B-oxidation of fatty acids. Central to these effects is the alteration of a-ketoglutarate (KG) homeostasis. In Al-exposed cells, KG is preferentially used to quench ROS leading to succinate accumulation and HIF-1a stabilization. Moreover, the channeling of KG to combat oxidative stress leads to a reduction of L-carnitine biosynthesis and a concomitant decrease in fatty acid oxidation. The fluidity and interaction of these mtabolic modules and the implications of these findings in liver-related disorders are discussed herein. (citation) (Note: The importance of these findings is relevant especially for brain development and for chronic autoimmune disorders such as diabetes and autism. The disruption of mitochondrial metabolism is significant in the increased risk of vaccine-injury in persons who have underlying mitochondrial disorders – See the Hannah Poling case for more information.) http://vaxtruth.org/2011/08/vaccines-mitochondrial/

DS doesn't like it when someone young can get the truth across to others. I put nothing past them.